Association between activated K-ras and c-erbB-2 oncogenes with “high-risk” and “low-risk” Human Papilloma Virus types in preinvasive cervical lesions
- 21 August 2000
- journal article
- Published by Elsevier in Mutation Research - Genetic Toxicology and Environmental Mutagenesis
- Vol. 469 (1), 127-134
- https://doi.org/10.1016/s1383-5718(00)00066-8
Abstract
No abstract availableKeywords
This publication has 16 references indexed in Scilit:
- Cancer: The Emerging Molecular BiologyHospital Practice, 2000
- Epidemiologic Evidence and Human Papillomavirus Infection as a Necessary Cause of Cervical CancerJNCI Journal of the National Cancer Institute, 1999
- Prognostic Significance of p34cdc2Cyclin-Dependent Kinase and MIB1 Overexpression, and HER-2/neuGene Amplification Detected by Fluorescence In Situ Hybridization in Breast CancerAmerican Journal of Clinical Pathology, 1999
- Frequent Amplification of C-erbB2 (HER-2/Neu) Oncogene in Cervical Carcinoma as Detected by Non-Fluorescence in situ Hybridization Technique on Paraffin SectionsOncology, 1999
- K-ras point mutation occurs in the early stage of carcinogenesis in lung cancerBritish Journal of Cancer, 1998
- Identification of H, K, and N-rasPoint Mutations in Stage IB Cervical CarcinomaGynecologic Oncology, 1997
- Prevalence of Human Papillomavirus in Cervical Cancer: a Worldwide PerspectiveJNCI Journal of the National Cancer Institute, 1995
- Analysis and clinical implications of k‐ras gene mutations and infection with human papillomavirus types 16 and 18 in primary adenocarcinoma of the uterine cervixInternational Journal of Cancer, 1995
- Estimates of the worldwide incidence of eighteen major cancers in 1985International Journal of Cancer, 1993
- The multistep nature of cancerTrends in Genetics, 1993