Effects of atrial natriuretic factor, sodium nitroprusside, and acetylcholine on cyclic GMP levels and relaxation in rat aorta
- 1 September 1985
- journal article
- research article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 115 (2-3), 219-229
- https://doi.org/10.1016/0014-2999(85)90694-6
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Effects of Na+, K+-pump inhibitors and membrane depolarizing agents on acetylcholine-induced endothelium-dependent relaxation and cyclic GMP accumulation in rat aortaEuropean Journal of Pharmacology, 1985
- Effect of native and synthetic atrial natriuretic factor on cyclic GMPBiochemical and Biophysical Research Communications, 1984
- Effect of cyanide on nitrovasodilator-induced relaxation, cyclic GMP accumulation and guanylate cyclase activation in rat aortaEuropean Journal of Pharmacology, 1984
- Vasodilator profile of synthetic atrial natriuretic factorEuropean Journal of Pharmacology, 1984
- Cardionatrin causes vasodilation in vitro which is not dependent on the presence of endothelial cellsEuropean Journal of Pharmacology, 1984
- Atrial natriuretic factor. Characterization and partial purification.Hypertension, 1983
- Agonist-induced endothelium-dependent relaxation in rat thoracic aorta may be mediated through cGMP.Circulation Research, 1983
- Soluble guanylate cyclase purified from bovine lung contains heme and copperFEBS Letters, 1981
- The Electrogenic NA+,K+-Pump in Smooth Muscle: Physiologic and Pharmacologic SignificanceAnnual Review of Pharmacology and Toxicology, 1980
- Sodium nitroprusside and other smooth muscle-relaxants increase cyclic GMP levels in rat ductus deferensNature, 1977