Spontaneous firing as an epigenetic factor in brain development — physiological consequences of chronic tetrodotoxin and picrotoxin exposure on cultured rat neocortex neurons
- 1 January 1992
- journal article
- Published by Elsevier in Developmental Brain Research
- Vol. 65 (1), 57-64
- https://doi.org/10.1016/0165-3806(92)90008-k
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Chronic Suppression of Bioelectric Activity and Cell Survival in Primary Cultures of Rat Cerebral Cortex: Biochemical ObservationsEuropean Journal of Neuroscience, 1991
- Control of NMDA receptor-mediated activity by GABAergic mechanisms in mature and developing rat neocortexDevelopmental Brain Research, 1990
- Burst generating and regular spiking layer 5 pyramidal neurons of rat neocortex have different morphological featuresJournal of Comparative Neurology, 1990
- Spontaneous activity mediated by NMDA receptors in immature rat entorhinal cortex in vitroNeuroscience Letters, 1989
- Seizure-like activity and cellular damage in rat hippocampal neurons in cell cultureNeuron, 1989
- Activity-dependent regulation of GABA expression in the visual cortex of adult monkeysNeuron, 1988
- Accelerated neural network formation in rat cerebral cortex cultures chronically disinhibited with picrotoxinExperimental Neurology, 1987
- Synaptogenesis in rat cerebral cortex cultures is affected during chronic blockade of spontaneous bioelectric activity by tetrodotoxinDevelopmental Brain Research, 1985
- Patterns of spontaneous bioelectric activity during maturation in culture of fetal rodent medulla and spinal cord tissuesJournal of Neurobiology, 1972
- Maturation of cultured embryonic CNS tissues during chronic exposure to agents which prevent bioelectric activityBrain Research, 1968