Abstract
The eosinopenic response of humans to norepinephrine is much weaker than that to epinephrine. The marked fall of the circulating eosinophils observed during emotional stress and the accompanying cardiovascular manifestations (cardiac acceleration, palpitations, elevation of systolic but not of diastolic blood pressure) suggest that the neurovegetative reactions to emotional tension are prevailingly caused by a discharge of epinephrine and only to a minor extent, if at all, to norepinephrine.