Mitochondrial Import and Accumulation of α-Synuclein Impair Complex I in Human Dopaminergic Neuronal Cultures and Parkinson Disease Brain
Top Cited Papers
Open Access
- 1 April 2008
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 283 (14), 9089-9100
- https://doi.org/10.1074/jbc.m710012200
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- A heterozygous effect for PINK1 mutations in Parkinson's disease?Annals of Neurology, 2006
- Mitochondrial dysfunction and oxidative stress in neurodegenerative diseasesNature, 2006
- Chaperone-like activities of α-synuclein: α-Synuclein assists enzyme activities of esterasesBiochemical and Biophysical Research Communications, 2006
- Parkinson's Disease Brain Mitochondrial Complex I Has Oxidatively Damaged Subunits and Is Functionally Impaired and MisassembledJournal of Neuroscience, 2006
- Human mitochondrial complex I assembly is mediated by NDUFAF1The FEBS Journal, 2005
- DJ-1( PARK7), a novel gene for autosomal recessive, early onset parkinsonismNeurological Sciences, 2003
- Phosphorylation Enhances Mitochondrial Targeting of GSTA4-4 through Increased Affinity for Binding to Cytoplasmic Hsp70Journal of Biological Chemistry, 2003
- Mitochondrial respiratory chain function in multiple system atrophyMovement Disorders, 1997
- Mitochondrial Complex I Deficiency in Parkinson's DiseaseJournal of Neurochemistry, 1990
- Point mutations define a sequence flanking the AUG initiator codon that modulates translation by eukaryotic ribosomesCell, 1986