Chrysamine-G, a lipophilic analogue of congo red, inhibits Aβ-induced toxicity in PC12 cells
- 1 October 1998
- journal article
- Published by Elsevier in Life Sciences
- Vol. 63 (20), 1807-1814
- https://doi.org/10.1016/s0024-3205(98)00454-8
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Impairment of Glucose and Glutamate Transport and Induction of Mitochondrial Oxidative Stress and Dysfunction in Synaptosomes by Amyloid β‐Peptide: Role of the Lipid Peroxidation Product 4‐HydroxynonenalJournal of Neurochemistry, 1997
- Morphology and Toxicity of Aβ-(1-42) Dimer Derived from Neuritic and Vascular Amyloid Deposits of Alzheimer's DiseaseJournal of Biological Chemistry, 1996
- RAGE and amyloid-β peptide neurotoxicity in Alzheimer's diseaseNature, 1996
- Direct evidence of oxidative injury produced by the Alzheimer's β-Amyloid peptide (1–40) in cultured hippocampal neuronsExperimental Neurology, 1995
- Rifampicin Prevents the Aggregation and Neurotoxicity of Amyloid β Protein in VitroBiochemical and Biophysical Research Communications, 1994
- β-Amyloid Peptide Free Radical Fragments Initiate Synaptosomal Lipoperoxidation in a Sequence-Specific Fashion: Implications to Alzheimer′s DiseaseBiochemical and Biophysical Research Communications, 1994
- Calcium-destabilizing and neurodegenerative effects of aggregated β-amyloid peptide are attenuated by basic FGFBrain Research, 1993
- Complement activation and β-amyloid-mediated neurotoxicity in Alzheimer's diseaseResearch in Immunology, 1992
- Vitamin E protects nerve cells from amyloid βprotein toxicityBiochemical and Biophysical Research Communications, 1992
- Neurotrophic and Neurotoxic Effects of Amyloid β Protein: Reversal by Tachykinin NeuropeptidesScience, 1990