Overexpression of SOD1 protects vulnerable motor neurons after spinal cord injury by attenuating mitochondrial cytochrome c release
Open Access
- 4 October 2002
- journal article
- fj express-summaries
- Published by Wiley in The FASEB Journal
- Vol. 16 (14), 1997-1999
- https://doi.org/10.1096/fj.02-0251fje
Abstract
Defective Cu, Zn-superoxide dismutase (SOD1) is responsible for some types of amyotrophic lateral sclerosis, and ventral horn motor neurons (VMN) have been shown to die through a mitochondria-depende...Keywords
Funding Information
- National Institutes of Health (P50 NS14543, RO1 NS25372, RO1 NS36147, PO1 NS37520, RO1 NS38653, NO1 NS82386)
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