Overexpression of SOD1 protects vulnerable motor neurons after spinal cord injury by attenuating mitochondrial cytochrome c release

Abstract
Defective Cu, Zn-superoxide dismutase (SOD1) is responsible for some types of amyotrophic lateral sclerosis, and ventral horn motor neurons (VMN) have been shown to die through a mitochondria-depende...
Funding Information
  • National Institutes of Health (P50 NS14543, RO1 NS25372, RO1 NS36147, PO1 NS37520, RO1 NS38653, NO1 NS82386)