INCREASE IN MYOCARDIAL Gi-PROTEINS IN HEART FAILURE
- 1 October 1988
- journal article
- research article
- Published by Elsevier in The Lancet
- Vol. 332 (8617), 936-937
- https://doi.org/10.1016/s0140-6736(88)92601-3
Abstract
No abstract availableThis publication has 9 references indexed in Scilit:
- Effects of α- and β-adrenergic agonists, phosphodiesterase inhibitors and adenosine on isolated human heart muscle preparationsTrends in Pharmacological Sciences, 1987
- Deficient production of cyclic AMP: pharmacologic evidence of an important cause of contractile dysfunction in patients with end-stage heart failure.Circulation, 1987
- Reduction in the activity of the stimulatory guanine nucleotide-binding protein in the myocardium of spontaneously hypertensive ratsJournal of Molecular and Cellular Cardiology, 1987
- The positive inotropic response to milrinone in isolated human and guinea pig myocardiumNaunyn-Schmiedebergs Archiv für experimentelle Pathologie und Pharmakologie, 1986
- Two guanine nucleotide-binding proteins in rat brain serving as the specific substrate of islet-activating protein, pertussis toxin. Interaction of the alpha-subunits with beta gamma-subunits in development of their biological activities.Journal of Biological Chemistry, 1986
- Dual regulation of adenylate cyclase. A signal transduction mechanism of membrane receptorsBasic Research in Cardiology, 1986
- $beta;-Adrenergic function in heart muscle disease and heart failureJournal of Molecular and Cellular Cardiology, 1985
- Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4Nature, 1970