MECHANISM OF N-HYDROXYACETYLARYLAMINE MUTAGENICITY IN THE SALMONELLA TEST SYSTEM - METABOLIC-ACTIVATION OF N-HYDROXYPHENACETIN BY LIVER AND KIDNEY FRACTIONS FROM RAT, MOUSE, HAMSTER, AND MAN
- 1 January 1980
- journal article
- research article
- Vol. 18 (1), 117-127
Abstract
No abstract availableThis publication has 10 references indexed in Scilit:
- Ascorbic acid inhibits covalent binding of enzymatically generated 2-acetyl-aminofluorene-N-sulfate to DNA under conditions in which it increases mutagenesis in salmonella TA-1538Life Sciences, 1979
- Synthesis of N-hydroxyacetaminophen, a postulated toxic metabolite of acetaminophen, and its phenolic sulfate conjugateJournal of Medicinal Chemistry, 1978
- MUTAGENIC ACTIVATION OF N-HYDROXY-2-ACETYLAMINOFLUORENE IN SALMONELLA TEST SYSTEM - ROLE OF DEACETYLATION BY LIVER AND KIDNEY FRACTIONS FROM MOUSE AND RAT1978
- MECHANISMS OF INHIBITION BY ASCORBATE OF MICROBIAL MUTAGENESIS INDUCED BY N-NITROSO COMPOUNDS1978
- MECHANISM OF INVITRO MUTAGENIC ACTIVATION AND COVALENT BINDING OF N-HYDROXY-2-ACETYLAMINOFLUORENE IN ISOLATED LIVER-CELL NUCLEI FROM RAT AND MOUSE1978
- Role of sulfotransferase from rat liver in the mutagenicity of N-hydroxy-2-acetylaminofluorene in Salmonella typhimuriumBiochemical Pharmacology, 1977
- N-HYDROXYLATION OF PHENACETIN BY HAMSTER LIVER-MICROSOMES1976
- Neoplasia in the rat induced by N-Hydroxyphenacetin, a metabolite of phenacetinPathology, 1976
- ENZYMATIC DEACETYLATION OF N-HYDROXY-2-ACETYLAMINOFLUORENE BY LIVER MICROSOMES1966
- ENZYMATIC REDUCTION OF N-HYDROXY DERIVATIVES OF 2-ACETYLAMINOFLUORENE AND RELATED CARCINOGENS BY TISSUE PREPARATIONS1965