Exercise training increases acetylcholine-stimulated endothelium-derived nitric oxide release in spontaneously hypertensive rats
- 1 November 1996
- journal article
- Published by Springer Nature in Journal of Biomedical Science
- Vol. 3 (6), 454-460
- https://doi.org/10.1007/bf02258049
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Physical conditioning decreases norepinephrine-induced vasoconstriction in rabbits. Possible roles of norepinephrine-evoked endothelium-derived relaxing factor.Circulation, 1994
- Chronic exercise in dogs increases coronary vascular nitric oxide production and endothelial cell nitric oxide synthase gene expression.Circulation Research, 1994
- Physical conditioning can modulate endothelium-dependent vasorelaxation in rabbits.Arteriosclerosis and Thrombosis: A Journal of Vascular Biology, 1993
- Renovascular hypertension impairs formation of endothelium‐derived relaxing factors and sensitivity to endothelin‐1 in resistance arteriesBritish Journal of Pharmacology, 1991
- Effects of native and oxidized low density lipoproteins on formation and inactivation of endothelium-derived relaxing factor.Arteriosclerosis and Thrombosis: A Journal of Vascular Biology, 1991
- Indirect evidence for release of endothelium-derived relaxing factor in human forearm circulation in vivo. Blunted response in essential hypertension.Circulation, 1990
- Low-density lipoproteins inhibit endothelium-dependent relaxation in rabbit aortaNature, 1987
- Decreased responsiveness of the aortae of hypertensive rats to acetylcholine, histamine and noradrenalineBritish Journal of Pharmacology, 1987
- Endothelium-dependent contractions to acetylcholine in the aorta of the spontaneously hypertensive rat.Hypertension, 1986
- Effect of aerobic exercise training on patients with systemic arterial hypertensionThe American Journal of Medicine, 1984