Effects of caspase inhibition on the progression of experimental glomerulonephritis
- 1 June 2003
- journal article
- Published by Elsevier in Kidney International
- Vol. 63 (6), 2050-2064
- https://doi.org/10.1046/j.1523-1755.2003.00000.x
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- Role and regulation of activation of caspases in cisplatin-induced injury to renal tubular epithelial cellsKidney International, 2001
- Interferon-γ inhibits experimental renal fibrosisKidney International, 1999
- Distinct interleukin-1β-converting enzyme family proteases mediate cisplatin- and staurosporine-induced apoptosis of mouse proximal tubule cellsLife Sciences, 1998
- The caspase family of cysteine proteasesBritish Medical Bulletin, 1997
- CPP32/Apopain Is a Key Interleukin 1β Converting Enzyme-like Protease Involved in Fas-mediated ApoptosisJournal of Biological Chemistry, 1996
- Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosisNature, 1995
- Requirement of an ICE/CED-3 protease for Fas/APO-1-mediated apoptosisNature, 1995
- Altered Cytokine Export and Apoptosis in Mice Deficient in Interleukin-1β Converting EnzymeScience, 1995
- Mesangial cell apoptosis: the major mechanism for resolution of glomerular hypercellularity in experimental mesangial proliferative nephritis.Journal of Clinical Investigation, 1994
- Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation.The Journal of cell biology, 1992