A1 Functions at the Mitochondria to Delay Endothelial Apoptosis in Response to Tumor Necrosis Factor
Open Access
- 1 June 2000
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 275 (24), 18099-18107
- https://doi.org/10.1074/jbc.m908925199
Abstract
No abstract availableKeywords
This publication has 58 references indexed in Scilit:
- NF-κB Activation Is Required for Human Endothelial Survival during Exposure to Tumor Necrosis Factor-α but Not to Interleukin-1β or LipopolysaccharidePublished by Elsevier ,1999
- The prosurvival Bcl-2 homolog Bfl-1/A1 is a direct transcriptional target of NF-kappa B that blocks TNFalpha -induced apoptosisGenes & Development, 1999
- ACTIVATION OF INTERLEUKIN-1β-CONVERTING ENZYME BY NIGERICIN IS INDEPENDENT OF APOPTOSISCytokine, 1998
- IEX -1L, an Apoptosis Inhibitor Involved in NF-κB-Mediated Cell SurvivalScience, 1998
- BCL-2 FAMILY: Regulators of Cell DeathAnnual Review of Immunology, 1998
- Cytokine Induced Expression of Porcine Inhibitor of Apoptosis Protein (iap) Family Member Is Regulated by NF-κBBiochemical and Biophysical Research Communications, 1998
- Bcl-xL Functions Downstream of Caspase-8 to Inhibit Fas- and Tumor Necrosis Factor Receptor 1-induced Apoptosis of MCF7 Breast Carcinoma CellsPublished by Elsevier ,1998
- FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling ComplexCell, 1996
- Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell DeathCell, 1996
- Altered Cytokine Export and Apoptosis in Mice Deficient in Interleukin-1β Converting EnzymeScience, 1995