Hereditary angioedema caused by missense mutations in the factor XII gene: Clinical features, trigger factors, and therapy
- 1 July 2009
- journal article
- Published by Elsevier in Journal of Allergy and Clinical Immunology
- Vol. 124 (1), 129-134
- https://doi.org/10.1016/j.jaci.2009.03.038
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Plasma biomarkers of acute attacks in patients with angioedema due to C1‐inhibitor deficiencyAllergy, 2009
- Oestrogen‐dependent hereditary angio‐oedema with normal C1 inhibitor: description of six new cases and review of pathogenic mechanisms and treatmentAllergy, 2008
- Potential role of vasomotor effects of fibrinogen in bradykinin-induced angioedemaJournal of Allergy and Clinical Immunology, 2008
- Benefits and risks of danazol in hereditary angioedema: a long-term survey of 118 patientsAnnals of Allergy, Asthma & Immunology, 2008
- Increased Activity of Coagulation Factor XII (Hageman Factor) Causes Hereditary Angioedema Type IIIAmerican Journal of Human Genetics, 2006
- Missense mutations in the coagulation factor XII (Hageman factor) gene in hereditary angioedema with normal C1 inhibitorBiochemical and Biophysical Research Communications, 2006
- Hereditary Angioedema: New Findings Concerning Symptoms, Affected Organs, and CourseThe American Journal of Medicine, 2006
- Hereditary angio-oedema: effective treatment with the progestogen-only pill in a young womanBritish Journal of Dermatology, 2004
- Efficacy of danazol treatment in a patient with the new variant of hereditary angio-oedema (HAE III)British Journal of Dermatology, 2004
- Clinical, biochemical, and genetic characterization of a novel estrogen-dependent inherited form of angioedemaJournal of Allergy and Clinical Immunology, 2000