Dystrophin glycoprotein complex dysfunction: A regulatory link between muscular dystrophy and cancer cachexia
- 1 November 2005
- journal article
- Published by Elsevier in Cancer Cell
- Vol. 8 (5), 421-432
- https://doi.org/10.1016/j.ccr.2005.10.004
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- Adenocarcinoma of oesophagus: what exactly is the size of the problem and who is at risk?Gut, 2005
- Molecular Mechanisms of Muscle AtrophyCell, 2004
- Aberrant expression, processing and degradation of dystroglycan in squamous cell carcinomasEuropean Journal Of Cancer, 2004
- The IGF-1/PI3K/Akt Pathway Prevents Expression of Muscle Atrophy-Induced Ubiquitin Ligases by Inhibiting FOXO Transcription FactorsMolecular Cell, 2004
- Unlike insulin, amino acids stimulate p70S6Kbut not GSK-3 or glycogen synthase in human skeletal muscleAmerican Journal of Physiology-Endocrinology and Metabolism, 2004
- Cancer cachexia: the molecular mechanismsThe International Journal of Biochemistry & Cell Biology, 2003
- Signalling pathways that mediate skeletal muscle hypertrophy and atrophyNature Cell Biology, 2003
- Cachexia in cancer patientsNature Reviews Cancer, 2002
- Post-translational disruption of dystroglycan–ligand interactions in congenital muscular dystrophiesNature, 2002
- Why do patients with weight loss have a worse outcome when undergoing chemotherapy for gastrointestinal malignancies?European Journal Of Cancer, 1998