FcγRI (CD64) Contributes Substantially to Severity of Arthritis, Hypersensitivity Responses, and Protection from Bacterial Infection
Open Access
- 1 March 2002
- Vol. 16 (3), 391-402
- https://doi.org/10.1016/s1074-7613(02)00294-7
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- IgG Fc ReceptorsAnnual Review of Immunology, 2001
- Exacerbation of Autoantibody-Mediated Hemolytic Anemia by Viral InfectionJournal of Virology, 2000
- Evidence for an intracellular niche forBordetella pertussisin broncho-alveolar lavage cells of miceFEMS Immunology & Medical Microbiology, 1999
- Evidence for an intracellular niche for Bordetella pertussis in broncho-alveolar lavage cells of miceFEMS Immunology & Medical Microbiology, 1999
- Impaired IgG-Dependent Anaphylaxis and Arthus Reaction in FcγRIII (CD16) Deficient MiceImmunity, 1996
- Augmented humoral and anaphylactic responses in FcγRII-deficient miceNature, 1996
- Efficient presentation of soluble antigen by cultured human dendritic cells is maintained by granulocyte/macrophage colony-stimulating factor plus interleukin 4 and downregulated by tumor necrosis factor alpha.The Journal of Experimental Medicine, 1994
- FcR γ chain deletion results in pleiotrophic effector cell defectsCell, 1994
- Fc ReceptorsAnnual Review of Immunology, 1991
- Monoclonal anti-erythrocyte autoantibodies derived from NZB mice cause autoimmune hemolytic anemia by two distinct pathogenic mechanismsInternational Immunology, 1990