Angiotensin 1–7 increases quantal content and facilitation at the frog neuromuscular junction
- 15 February 2002
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 927 (2), 208-211
- https://doi.org/10.1016/s0006-8993(01)03357-1
Abstract
No abstract availableKeywords
This publication has 14 references indexed in Scilit:
- Prejunctional angiotensin receptors involved in the facilitation of noradrenaline release in mouse tissuesBritish Journal of Pharmacology, 1999
- Calcitonin gene‐related peptide acts presynaptically to increase quantal size and output at frog neuromuscular junctionsThe Journal of Physiology, 1998
- Effects of angiotensin II and angiotensin-(1-7) on the release of [3H]norepinephrine from rat atria.Hypertension, 1994
- Regulatory role of brain angiotensins in the control of physiological and behavioral responsesBrain Research Reviews, 1992
- Angiotensin-(1-7). A member of circulating angiotensin peptides.Hypertension, 1991
- An alternative strategy for the radioimmunoassay of angiotensin peptides using amino-terminal-directed antisera: measurement of eight angiotensin peptides in human plasmaJournal Of Hypertension, 1990
- Vasopressin produces long-lasting increase in transmitter releaseBrain Research, 1990
- Catecholamines, insulin and ACTH increase quantal size at the frog neuromuscular junctionBrain Research, 1986
- Actions of polypeptides at the neuromuscular junctionInflammation Research, 1985
- Adrenocorticotropic Hormone Causes Long-Lasting Potentiation of Transmitter Release from Frog Motor Nerve TerminalsScience, 1983