RESPIRATORY TRACT DAMAGE IN BURNS: PATHOPHYSIOLOGY AND THERAPY*
- 1 August 1968
- journal article
- Published by Wiley in Annals of the New York Academy of Sciences
- Vol. 150 (3), 618-626
- https://doi.org/10.1111/j.1749-6632.1968.tb14714.x
Abstract
Summary: (1) Of the 27 burn fatalities with respiratory tract involvement, this internal damage was the primary cause of death in 20 cases.(2) The edema and sodium contents in the lungs become maximal in 12 to 48 hr following the burn.(3) The surface tension in the lungs is abnormally elevated in respiratory burns and may be one of the causes of nonobstructive atelectasis.(4) The venous clotting time becomes depressed in the acute phase of respiratory burn and returns to normal in 24 hr.(5) Lymph drainage into the left thoracic duct vestibule decreases during the acute phase of the burn in the dog.(6) In the acute phase of the burn, the respiratory insufficiency is secondary to edema and to patchy atelectasis which can be reduced by automatic positive pressure respiration.(7) Infection of pulmonary and tracheobronchial tree plays a major role after 48 hr and early antibiotic coverage may help to control it.This publication has 7 references indexed in Scilit:
- Metabolic acidosis in burns.BMJ, 1968
- Cause of Death from BurnsAnnals of Surgery, 1965
- Burn TherapyAnnals of Surgery, 1962
- Burn Therapy II. The Revelation of Respiratory Tract Damage as a Principal Killer of the Burned PatientAnnals of Surgery, 1962
- Burn Therapy*Annals of Surgery, 1960
- Respiratory Burns with Special Reference to Pulmonary Edema and CongestionCirculation, 1952
- EFFECTS OF PLASMA AND FLUID ON PULMONARY COMPLICATIONS IN BURNED PATIENTSArchives of Internal Medicine, 1946