Double-Strand Break Repair-Independent Role for BRCA2 in Blocking Stalled Replication Fork Degradation by MRE11
Top Cited Papers
- 1 May 2011
- Vol. 145 (4), 529-542
- https://doi.org/10.1016/j.cell.2011.03.041
Abstract
No abstract availableKeywords
This publication has 61 references indexed in Scilit:
- Purified human BRCA2 stimulates RAD51-mediated recombinationNature, 2010
- Mitotic homologous recombination maintains genomic stability and suppresses tumorigenesisNature Reviews Molecular Cell Biology, 2010
- Hydroxyurea-Stalled Replication Forks Become Progressively Inactivated and Require Two Different RAD51-Mediated Pathways for Restart and RepairMolecular Cell, 2010
- Brh2 Promotes a Template-Switching Reaction Enabling Recombinational Bypass of Lesions during DNA SynthesisMolecular Cell, 2009
- The Carboxyl Terminus of Brca2 Links the Disassembly of Rad51 Complexes to Mitotic EntryCurrent Biology, 2009
- DNA end resection: Many nucleases make light workDNA Repair, 2009
- Mre11 Dimers Coordinate DNA End Bridging and Nuclease Processing in Double-Strand-Break RepairCell, 2008
- Secondary mutations as a mechanism of cisplatin resistance in BRCA2-mutated cancersNature, 2008
- A forward chemical genetic screen reveals an inhibitor of the Mre11–Rad50–Nbs1 complexNature Chemical Biology, 2008
- Minding the gap: The underground functions of BRCA1 and BRCA2 at stalled replication forksDNA Repair, 2007