The syndrome of delayed traumatic intracerebral hemorrhage, ortraumatische Spätapoplexie, was first described in 1891 by Bollinger,1who stated that even a mild injury to the head might be followed by the development of foci of softening in the cerebrum or medulla, alterations in the blood vessel walls and intracranial hemorrhage. The syndrome has not aroused a great deal of interest during the fifty years that have elapsed, but there have been occasional reports of the onset of hemiplegia or other evidences of cerebral damage days, or even weeks, after what appeared to have been a slight injury to the head. In spite of some controversy, both with respect to the exact mechanism of the hemorrhage and its relationship to preexisting vascular disease, delayed traumatic apoplexy has come to be regarded as a disease entity. The symptoms may resemble those of ordinary cerebral hemorrhage, embolism or thrombosis or those