High glucose induces MCP-1 expression partly via tyrosine kinase–AP-1 pathway in peritoneal mesothelial cells
- 1 July 2001
- journal article
- Published by Elsevier in Kidney International
- Vol. 60 (1), 55-64
- https://doi.org/10.1046/j.1523-1755.2001.00770.x
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Angiotensin II Signaling in Vascular Smooth Muscle Cells Under High Glucose ConditionsHypertension, 1999
- Leukocyte migration across human peritoneal mesothelial cells is dependent on directed chemokine secretion and ICAM-1 expressionKidney International, 1998
- Hyperosmolality suppresses but TGFβ1 increases MMP9 in human peritoneal mesothelial cellsKidney International, 1997
- Human peritoneal fibroblast proliferation in 3-dimensional culture: Modulation by cytokines, growth factors and peritoneal dialysis effluentKidney International, 1997
- Tyrosine Kinase Inhibitors, Genistein and Herbimycin A, Do Not Block Interleukin-1 β-Induced Activation of NF-κB in Rat Mesangial CellsBiochemical and Biophysical Research Communications, 1996
- Toxicity of osmotic solutes on human mesothelial cells in vitroKidney International, 1992
- Effects of high glucose on cellular proliferation and fibronectin production by cultured human mesangial cellsKidney International, 1992
- Structure of human monocyte chemotactic protein gene and its regulation by TPABiochemical and Biophysical Research Communications, 1990
- Human monocyte chemoattractant protein‐1 (MCP‐1) Full‐length cDNA cloning, expression in mitogen‐stimulated blood mononuclear leukocytes, and sequence similarity to mouse competence gene JEFEBS Letters, 1989
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976