Diverse Targets of the Transcription Factor STAT3 Contribute to T Cell Pathogenicity and Homeostasis
Top Cited Papers
- 20 May 2010
- Vol. 32 (5), 605-615
- https://doi.org/10.1016/j.immuni.2010.05.003
Abstract
No abstract availableKeywords
Funding Information
- National Institute of Arthritis and Musculoskeletal and Skin Diseases
- Wellcome Trust
- National Institutes of Health (1 K22 AR53953-01)
This publication has 58 references indexed in Scilit:
- A protective function for interleukin 17A in T cell–mediated intestinal inflammationNature Immunology, 2009
- The interleukin 23 receptor is essential for the terminal differentiation of interleukin 17–producing effector T helper cells in vivoNature Immunology, 2009
- Global Mapping of H3K4me3 and H3K27me3 Reveals Specificity and Plasticity in Lineage Fate Determination of Differentiating CD4+ T CellsImmunity, 2009
- The costimulatory molecule ICOS regulates the expression of c-Maf and IL-21 in the development of follicular T helper cells and TH-17 cellsNature Immunology, 2008
- Molecular Antagonism and Plasticity of Regulatory and Inflammatory T Cell ProgramsImmunity, 2008
- Interleukin-23 Restrains Regulatory T Cell Activity to Drive T Cell-Dependent ColitisImmunity, 2008
- Impaired TH17 cell differentiation in subjects with autosomal dominant hyper-IgE syndromeNature, 2008
- T Helper 17 Lineage Differentiation Is Programmed by Orphan Nuclear Receptors RORα and RORγImmunity, 2008
- Chromatin Modifications and Their FunctionCell, 2007
- Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cellsNature, 2006