Long Uninterrupted CGG Repeats within the First Exon of the Human FMR1 Gene Are Not Intrinsically Unstable in Transgenic Mice
Open Access
- 1 June 1998
- Vol. 50 (2), 229-240
- https://doi.org/10.1006/geno.1998.5299
Abstract
No abstract availableKeywords
This publication has 57 references indexed in Scilit:
- FMR1 Premutation Allele (CGG)(81) Is Stable in MiceEuropean Journal of Human Genetics, 1997
- Anomalous Rapid Electrophoretic Mobility of DNA Containing Triplet Repeats Associated with Human Disease GenesBiochemistry, 1995
- TRINUCLEOTIDE REPEAT EXPANSION AND HUMAN DISEASEAnnual Review of Genetics, 1995
- Population survey of the human FMR1 CGG repeat substructure suggests biased polarity for the loss of AGG interruptionsHuman Molecular Genetics, 1995
- SCA1 transgenic mice: A model for neurodegeneration caused by an expanded CAG trinucleotide repeatCell, 1995
- Hairpins are formed by the single DNA strands of the fragile X triplet repeats: structure and biological implications.Proceedings of the National Academy of Sciences, 1995
- Stability of an expanded trinucleotide repeat in the androgen receptor gene in transgenic miceNature Genetics, 1995
- Length of uninterrupted CGG repeats determines instability in the FMR1 geneNature Genetics, 1994
- Factors affecting the efficiency of introducing foreign DNA into mice by microinjecting eggs.Proceedings of the National Academy of Sciences, 1985
- Folic acid sensitive fragile sites are not limited to the human karyotype. Demonstration of nonrandom gaps and breaks in the Persian vole Ellobius lutescens Th. inducible by methotrexate, fluorodeoxyuridine, and aphidicolinHuman Genetics, 1985