Bcl-2-family proteins and the role of mitochondria in apoptosis
Top Cited Papers
- 16 October 2003
- journal article
- review article
- Published by Elsevier in Current Opinion in Cell Biology
- Vol. 15 (6), 691-699
- https://doi.org/10.1016/j.ceb.2003.10.004
Abstract
No abstract availableKeywords
This publication has 94 references indexed in Scilit:
- BCL-2 Selectively Interacts with the BID-induced Open Conformer of BAK, Inhibiting BAK Auto-oligomerizationJournal of Biological Chemistry, 2003
- Omi/HtrA2 catalytic cleavage of inhibitor of apoptosis (IAP) irreversibly inactivates IAPs and facilitates caspase activity in apoptosisGenes & Development, 2003
- BAX and BAK Regulation of Endoplasmic Reticulum Ca 2+ : A Control Point for ApoptosisScience, 2003
- Characterization of a Novel and Specific Inhibitor for the Pro-apoptotic Protease Omi/HtrA2Journal of Biological Chemistry, 2003
- Bax-type Apoptotic Proteins Porate Pure Lipid Bilayers through a Mechanism Sensitive to Intrinsic Monolayer CurvatureJournal of Biological Chemistry, 2002
- Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and DeathScience, 2001
- Preservation of Mitochondrial Structure and Function after Bid- or Bax-Mediated Cytochrome c ReleaseThe Journal of cell biology, 2000
- The Cell Death-promoting Gene DP5, Which Interacts with the BCL2 Family, Is Induced during Neuronal Apoptosis Following Exposure to Amyloid β ProteinPublished by Elsevier ,1999
- Inhibition of Bax Channel-Forming Activity by Bcl-2Science, 1997
- Bcl-xL forms an ion channel in synthetic lipid membranesNature, 1997