• 1 February 1969
    • journal article
    • Vol. 97 (2), 550-6
Abstract
The molecular basis of transient repression of beta-galactosidase by glucose was examined. This repression acted only at the level of transcription. Apparently, it was not mediated by the I-gene product. Analysis of single cells in a culture subjected to transient repression showed that essentially all cells initially experienced repression and later became gradually resistant to repression.