Mutant lambda phage repressor with a specific defect in its positive control function.

Abstract
The .lambda. phage repressor is both a positive and a negative regulator of gene transcription. A mutant .lambda. phage repressor that has specifically lost its activator function is described. The mutant binds to the .lambda. phage operator sites and represses the .lambda. phage promoters PR and PL. It fails to stimulate transcription from the promoter PRM. The mutation lies in that portion of repressors the amino-terminal domain, that mediates stimulation of PRM. The mutation apparently has altered that region of repressor which, in the wild-type, contacts RNA polymerase to activate transcription from PRM.

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