Cooexpression of FTDP-17 tau and GSK-3β in transgenic mice induce tau polymerization and neurodegeneration
- 27 July 2005
- journal article
- research article
- Published by Elsevier BV in Neurobiology of Aging
- Vol. 27 (9), 1258-1268
- https://doi.org/10.1016/j.neurobiolaging.2005.06.010
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- Promotion of Hyperphosphorylation by Frontotemporal Dementia Tau MutationsJournal of Biological Chemistry, 2004
- Role of Tau Protein in Both Physiological and Pathological ConditionsPhysiological Reviews, 2004
- Modeling Tau Polymerization in Vitro: A Review and SynthesisBiochemistry, 2003
- Glycogen Synthase Kinase-3β Inhibitors Prevent Cellular Polyglutamine Toxicity Caused by the Huntington’s Disease MutationJournal of Biological Chemistry, 2002
- Lithium protects cultured neurons against β‐amyloid‐induced neurodegenerationFEBS Letters, 1999
- Transgenic Expression of the Shortest Human Tau Affects Its Compartmentalization and Its Phosphorylation as in the Pretangle Stage of Alzheimer's DiseaseThe American Journal of Pathology, 1999
- Selective Phosphorylation of Adult Tau Isoforms in Mature Hippocampal Neurons Exposed to Fibrillar AβMolecular and Cellular Neuroscience, 1997
- β-Amyloid fibrils induce tau phosphorylation and loss of microtubule bindingNeuron, 1995
- A sequence of cytoskeleton changes related to the formation of neurofibrillary tangles and neuropil threadsActa Neuropathologica, 1994
- The microtubule binding repeats of tau protein assemble into filaments like those found in Alzheimer's diseaseFEBS Letters, 1992