Loss or Inhibition of uPA or MMP-9 Attenuates LV Remodeling and Dysfunction after Acute Pressure Overload in Mice
- 31 January 2005
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 166 (1), 15-25
- https://doi.org/10.1016/s0002-9440(10)62228-6
Abstract
No abstract availableKeywords
This publication has 89 references indexed in Scilit:
- Attenuation of Oxidative Stress and Remodeling by Cardiac Inhibitor of Metalloproteinase Protein TransferCirculation, 2004
- Hypertensive Myocardial Fibrosis and Diastolic DysfunctionHypertension, 2004
- Myocardial remodeling after discrete radiofrequency injury: effects of tissue inhibitor of matrix metalloproteinase-1 gene deletionAmerican Journal of Physiology-Heart and Circulatory Physiology, 2004
- Selective Targeting and Timing of Matrix Metalloproteinase Inhibition in Post-Myocardial Infarction RemodelingCirculation, 2003
- The role of myocardial fibrillar collagen in ventricular remodeling and functionJournal of Cardiac Failure, 2002
- Effects of Matrix Metalloproteinase Inhibition on Ventricular Remodeling Due to Volume OverloadCirculation, 2002
- Selective Matrix Metalloproteinase Inhibition Reduces Left Ventricular Remodeling but Does Not Inhibit Angiogenesis After Myocardial InfarctionCirculation, 2002
- Disruption of the Plasminogen Gene in Mice Abolishes Wound Healing after Myocardial InfarctionThe American Journal of Pathology, 2000
- Effects of Gene Deletion of the Tissue Inhibitor of the Matrix Metalloproteinase-type 1 (TIMP-1) on Left Ventricular Geometry and Function in MiceJournal of Molecular and Cellular Cardiology, 2000
- Development of recombinant adenoviruses that drive high level expression of the human metalloproteinase-9 and tissue inhibitor of metalloproteinase-1 and -2 genes: Characterization of their infection into rabbit smooth muscle cells and human MCF-7 adenocarcinoma cellsMatrix Biology, 1996