Myofilament Degradation and Dysfunction of Human Cardiomyocytes in Fabry Disease
- 1 June 2008
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 172 (6), 1482-1490
- https://doi.org/10.2353/ajpath.2008.070576
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Fabry’s Disease CardiomyopathyJournal of the American College of Cardiology, 2006
- Cardiomyocyte Stiffness in Diastolic Heart FailureCirculation, 2005
- Alterations in Myofilament Function Contribute to Left Ventricular Dysfunction in Pigs Early After Myocardial InfarctionCirculation Research, 2004
- Prevalence of Fabry Disease in Female Patients With Late-Onset Hypertrophic CardiomyopathyCirculation, 2004
- Comparative in vitro Expression Study of Four Fabry Disease Causing Mutations at Glutamine 279 of the α-Galactosidase A ProteinHuman Heredity, 2004
- Fabry disease: molecular studies in Italian patients and X inactivation analysis in manifesting carriersJournal of Medical Genetics, 2003
- Early Detection of Fabry Cardiomyopathy by Tissue Doppler ImagingCirculation, 2003
- The mechanism of the force enhancement by mgADP under simulated isChaemic conditions in rat cardiac myocytesThe Journal of Physiology, 2002
- Force production in mechanically isolated cardiac myocytes from human ventricular muscle tissueCardiovascular Research, 1998
- Morphometry and GH/IGF‐1 Axis Deficiency May Identify a Form of Dilated Cardiomyopathy Which Is Corrected by Recombinant Human Growth Hormone (rHGH)aAnnals of the New York Academy of Sciences, 1995