THE ETIOLOGY OF HYPERTENSION DUE TO COMPLETE RENAL ISCHEMIA
Open Access
- 1 December 1940
- journal article
- research article
- Published by Rockefeller University Press in The Journal of Experimental Medicine
- Vol. 72 (6), 763-776
- https://doi.org/10.1084/jem.72.6.763
Abstract
1. Perfusates of totally ischemic kidneys of cats contain a pressor substance which is not present in the perfusates of normal kidneys, ischemic hind limbs, or ischemic gravid uteri.This publication has 9 references indexed in Scilit:
- THE LIBERATION OF RENIN BY PERFUSION OF KIDNEYS FOLLOWING REDUCTION OF PULSE PRESSUREThe Journal of Experimental Medicine, 1940
- The production of a pressor substance by the totally ischemic kidneyAmerican Heart Journal, 1940
- THE PRESSOR EFFECTS OF HOMOLOGOUS AND HETEROLOGOUS INJECTIONS OF HEATED KIDNEY EXTRACTSAmerican Journal of Physiology-Legacy Content, 1940
- A CRYSTALLINE PRESSOR SUBSTANCE (ANGIOTONIN) RESULTING FROM THE REACTION BETWEEN RENIN AND RENIN-ACTIVATORThe Journal of Experimental Medicine, 1940
- THE HEMODYNAMIC EFFECT OF THE DIOXANE DERIVATIVE 933 F ON TRAINED UNANESTHETIZED NORMAL AND RENAL HYPERTENSIVE DOGS AND ITS EFFECT ON THE PRESSOR ACTION OF RENINAmerican Journal of Physiology-Legacy Content, 1939
- PRESSOR SUBSTANCE IN THE CORTEX OF THE KIDNEYAmerican Journal of Physiology-Legacy Content, 1938
- THE RECOVERY OF AN ADRENALIN-LIKE SUBSTANCE FROM THE KIDNEYAmerican Journal of Physiology-Legacy Content, 1938
- STUDIES ON EXPERIMENTAL HYPERTENSIONThe Journal of Experimental Medicine, 1934
- Niere und Kreislauf1Skandinavisches Archiv Für Physiologie, 1898